Johnson MB*, Wang PP*, Atabay KD et al. Single-cell analysis reveals transcriptional heterogeneity of neural progenitors in human cortex. Nat Neurosci. 2015;18(5):637-646.
Kwan KY, Lam MMS, Johnson MB et al. Species-dependent posttranscriptional regulation of NOS1 by FMRP in the developing cerebral cortex. Cell. 2012;149(4):899-911.
Johnson MB*, Kawasawa YI*, Mason CE et al. Functional and evolutionary insights into human brain development through global transcriptome analysis. Neuron. 2009;62(4):494-509.
Matthew Johnson, Ph.D.
Matthew Johnson is a research scientist I in the Stanley Center for Psychiatric Research of the Broad Institute of MIT and Harvard. His work is focused on understanding the functions of genes involved in synapse development that contribute to risk for schizophrenia and other psychiatric diseases. He specifically investigates synaptic pruning, a normal developmental process of synapse elimination that occurs in the human cortex from childhood through early adulthood that is thought to be overactive in the brains of patients with schizophrenia. His research aims to uncover the links between genetic risk, excess synaptic pruning, and disrupted brain function using animal models as well as human cells and tissues.
Prior to joining the Broad Institute in 2017, Johnson completed postdoctoral training with Chris Walsh in the Division of Genetics and Genomics at Boston Children's Hospital, working on the developmental contributions of distinct stem cell populations to the complex structure of the cortex.
Johnson obtained his Ph.D. in neurobiology at Yale University in the lab of Nenad Sestan, and holds a bachelor’s degree in neuroscience from Brown University.
Contact Matthew Johnson via email at firstname.lastname@example.org.