Gaca AO, Gilmore MS. Killing of VRE Enterococcus faecalis by commensal strains: Evidence for evolution and accumulation of mobile elements in the absence of competition. Gut Microbes. 2016;7(1):90-96.
Gaca AO, Kudrin P, Colomer-Winter C, et al. From (p) ppGpp to (pp) pGpp: Characterization of Regulatory Effects of pGpp Synthesized by the Small Alarmone Synthetase of Enterococcus faecalis. J Bacteriol. 2015; 197; 18; 2908-2919.
Gaca AO, Kajfasz JK, Miller JH, et al. Basal levels of (p) ppGpp in Enterococcus faecalis: the magic beyond the stringent response. mBio. 2013;4 (5): e00646-13.
Anthony Gaca, Ph.D.
Anthony Gaca is a research scientist I in the Infectious Disease and Microbiome Program of the Broad Institute of MIT and Harvard. As part of the Microbial ’Omics Core (MOC), Gaca’s goal is to utilize next-generation sequencing technologies to advance research aimed at understanding how the bacteria that live in and on us interact with one another and with the host to promote health or disease. To this end, he is working to improve the efficiency and robustness of existing processes or adopt new technologies for the sequencing of bacterial genomes, complex microbial communities, and their associated transcriptional profiles.
Gaca joined the Broad Institute in August 2018 after completing his postdoctoral research at Harvard Medical School. His research involved using functional genomics to understand how commensal bacterial evolved to become antibiotic-resistant, hospital-adapted pathogens.
Gaca received his Ph.D. from the University of Rochester, where he studied the molecular details of stress responses that render bacterial pathogens inert to antibiotic killing. He also holds a B.S. in microbiology and molecular genetics from Michigan State University.
Contact Anthony Gaca via email at firstname.lastname@example.org.