1.
Sullivan SE, Liao M, Smith RV, et al. Candidate-based screening via gene modulation in human neurons and astrocytes implicates FERMT2 in Aβ and TAU proteostasis. Hum Mol Genet. 2019;28(5):718-735. doi:10.1093/hmg/ddy376.
1.
Mostafavi S, Gaiteri C, Sullivan SE, et al. A molecular network of the aging human brain provides insights into the pathology and cognitive decline of Alzheimer’s disease. Nat Neurosci. 2018;21(6):811-819. doi:10.1038/s41593-018-0154-9.
1.
Roostaei T, Felsky D, Nazeri A, et al. Genetic influence of plasma homocysteine on Alzheimer’s disease. Neurobiol Aging. 2018;62:243.e7-243.e14. doi:10.1016/j.neurobiolaging.2017.09.033.
1.
Sekiya M, Wang M, Fujisaki N, et al. Integrated biology approach reveals molecular and pathological interactions among Alzheimer’s Aβ42, Tau, TREM2, and TYROBP in Drosophila models. Genome Med. 2018;10(1):26. doi:10.1186/s13073-018-0530-9.
1.
Yu L, Petyuk VA, Gaiteri C, et al. Targeted brain proteomics uncover multiple pathways to Alzheimer’s dementia. Ann Neurol. 2018;84(1):78-88. doi:10.1002/ana.25266.
1.
Deming Y, Li Z, Kapoor M, et al. Genome-wide association study identifies four novel loci associated with Alzheimer’s endophenotypes and disease modifiers. Acta Neuropathol. 2017;133(5):839-856. doi:10.1007/s00401-017-1685-y.
1.
Chen R, Zhang J, Wu Y, et al. Monoacylglycerol lipase is a therapeutic target for Alzheimer’s disease. Cell Rep. 2012;2(5):1329-39. doi:10.1016/j.celrep.2012.09.030.
1.
Patrick E, Rajagopal S, Wong H-KA, et al. Dissecting the role of non-coding RNAs in the accumulation of amyloid and tau neuropathologies in Alzheimer’s disease. Mol Neurodegener. 2017;12(1):51. doi:10.1186/s13024-017-0191-y.
1.
Bis JC, Jian X, Kunkle BW, et al. Whole exome sequencing study identifies novel rare and common Alzheimer’s-Associated variants involved in immune response and transcriptional regulation. Mol Psychiatry. 2020;25(8):1859-1875. doi:10.1038/s41380-018-0112-7.
1.
Singer AC, Martorell AJ, Douglas M, et al. Noninvasive 40-Hz light flicker to recruit microglia and reduce amyloid beta load. Nat Protoc. 2018;13(8):1850-1868. doi:10.1038/s41596-018-0021-x.