Tomalka JA, Owings A, Galeas-Pena M, et al. Enhanced production of eicosanoids in plasma and activation of DNA damage pathways in PBMCs are correlated with the severity of ancestral COVID-19 infection. medRxiv : the preprint server for health sciences. 2023. doi:10.1101/2023.09.14.23295549.
He YJ, Meghani K, Caron M-C, et al. DYNLL1 binds to MRE11 to limit DNA end resection in BRCA1-deficient cells. Nature. 2018;563(7732):522-526. doi:10.1038/s41586-018-0670-5.
Sasanuma H, Tsuda M, Morimoto S, et al. BRCA1 ensures genome integrity by eliminating estrogen-induced pathological topoisomerase II-DNA complexes. Proc Natl Acad Sci U S A. 2018;115(45):E10642-E10651. doi:10.1073/pnas.1803177115.
Stewart C, Leshchiner I, Hess J, Getz G. Comment on "DNA damage is a pervasive cause of sequencing errors, directly confounding variant identification". Science. 2018;361(6409). doi:10.1126/science.aas9824.
Sadler JBA, Wenzel DM, Strohacker LK, et al. A cancer-associated polymorphism in ESCRT-III disrupts the abscission checkpoint and promotes genome instability. Proc Natl Acad Sci U S A. 2018;115(38):E8900-E8908. doi:10.1073/pnas.1805504115.
Linder SJ, Mostoslavsky R. Put Your Mark Where Your Damage Is: Acetyl-CoA Production by ACLY Promotes DNA Repair. Mol Cell. 2017;67(2):165-167. doi:10.1016/j.molcel.2017.07.006.
Nghiem P, Park PK, Ys Y- son K, Desai BN, Schreiber SL. ATR is not required for p53 activation but synergizes with p53 in the replication checkpoint. J Biol Chem. 2002;277(6):4428-34. doi:10.1074/jbc.M106113200.
Schmidt DR, Schreiber SL. Molecular association between ATR and two components of the nucleosome remodeling and deacetylating complex, HDAC2 and CHD4. Biochemistry. 1999;38(44):14711-7.