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  • Showing 1-6 of 6 Results
2016
Genome-wide association analyses identify new risk variants and the genetic architecture of amyotrophic lateral sclerosis.van Rheenen, W., Shatunov A., Dekker AM, McLaughlin RL, Diekstra FP, Pulit SL, van der Spek RA, Võsa U., de Jong S., Robinson MR, Yang J., Fogh I., van Doormaal PT, Tazelaar GH, Koppers M., Blokhuis AM, Sproviero W., Jones AR, Kenna KP, van Eijk KR, et al. Nature genetics, 2016/07/25, (2016) Read More / View Supplemental Materials
Abstract
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition).Klionsky, DJ, Abdelmohsen K., Abe A., Abedin MJ, Abeliovich H., Acevedo Arozena A., Adachi H., Adams CM, Adams PD, Adeli K., Adhihetty PJ, Adler SG, Agam G., Agarwal R., Aghi MK, Agnello M., Agostinis P., Aguilar PV, Aguirre-Ghiso J., Airoldi EM, et al. Autophagy, 2016/01/02, Volume 12, Issue 1, p.1-222, (2016) Read More / View Supplemental Materials
2015
Inactivation of HDAC1 or HDAC2 induces gamma globin expression without altering cell cycle or proliferation.Esrick, EB, McConkey M., Lin K., Frisbee A., and Ebert BL American journal of hematology, 2015/03/23, (2015) Read More / View Supplemental Materials
Abstract
2014
Pan-cancer genetic analysis identifies PARK2 as a master regulator of G1/S cyclins.Gong, Y., Zack TI, Morris LG, Lin K., Hukkelhoven E., Raheja R., Tan IL, Turcan S., Veeriah S., Meng S., Viale A., Schumacher SE, Palmedo P., Beroukhim R., and Chan TA Nature genetics, 2014/05/04, (2014) Read More / View Supplemental Materials
Abstract
2013
Genome-wide association analysis identifies 13 new risk loci for schizophrenia.Ripke, S., O'Dushlaine C., Chambert K., Moran JL, Kähler AK, Akterin S., Bergen SE, Collins AL, Crowley JJ, Fromer M., Kim Y., Lee SH, Magnusson PK, Sanchez N., Stahl EA, Williams S., Wray NR, Xia K., Bettella F., Borglum AD, et al. Nature genetics, 2013/10/01, Volume 45, Issue 10, p.1150-9, (2013) Read More / View Supplemental Materials
Abstract
2010
Chemical genetic strategy identifies histone deacetylase 1 (HDAC1) and HDAC2 as therapeutic targets in sickle cell disease.Bradner, JE, Mak R., Tanguturi SK, Mazitschek R., Haggarty SJ, Ross K., Chang CY, Bosco J., West N., Morse E., Lin K., Shen JP, Kwiatkowski NP, Gheldof N., Dekker J., DeAngelo DJ, Carr SA, Schreiber SL, Golub T. R., and Ebert BL Proceedings of the National Academy of Sciences of the United States of America, 2010/07/13, Volume 107, Issue 28, p.12617-22, (2010) Read More / View Supplemental Materials
Abstract
  • Showing 1-6 of 6 Results